Anticoagulants (such as coumadin, heparin, heparinoids, and enoxaparin) can produce a TE.

Anticonvalsants (such as Carbamazepine and valproic acid) have been reported to occasionally induce a TE. The suggested mechanism is altered or reduced zinc and selenium levels.

Antidepressants (such as amphetamines, imipramine, desipramine, fluoxetine, sertaline, and dixyrazine) can produce a TE. Lithium induces hypothyroidism and can result in TE and alopecia.

Antifungal drugs (such as fluconazole and ketoconazole in high doses) have induced a reversible TE.

Antiinflammatory drugs (such as chronic NSAIDs) have induced a reversible TE.

Antimitotic drugs used in cancer or as an immunosuppressive can produce an anagen effluvium (acute loss of 80 % scalp hair) and at lower dose can induce a TE.

Angiotensin-converting enzyme inhibitors (captopril and enalapril) can induce TE. The mechanism appears to be binding of zinc, and is reversible with zinc supplements.

B-blockers (such as systemic and topical ophthalmologic proparanol, and metroprolol) can induce a chronic TE and diffuse alopecia.

Estrogen antagonist (such as tamoxifen, SERMS [selected estrogen receptor modifiers], and botanical phytoestrogens) can induce a TE and diffuse alopecia. These are estrogen competitive receptor inhibitors that inhibit estrogen action on the dermal papilla.

Heavy metals such as gold have induced severe alopecia.

Minoxidil given orally can induce hirsutism, while when applied topically, it can initiate a TE which usually resolves in several months.

Statins to reduce cholesterol (such as clofibrfate, triparanol, and cholestyramine) can produce TE which can be dose-related.