Chapter 13 As a junior doctor, you will be expected to perform procedures on your patients safely. As you become more senior, the complexity of the procedures will increase and the supervision will decrease. Just remember that practice makes perfect and the more procedures you do the more confident you will become. This section is aimed to guide you through most procedures you will encounter as a foundation doctor. Becoming competent, safe and skilful in undertaking interventions to help patients is one of the great joys of medicine. In lots of hospitals, two to four come made up in an ABG packet. (See Figs. 13.1 and 13.2.) Normal ABG values are shown in Table 13.1. Table 13.1 Normal ABG values. First ask yourself: does the patient have abnormally low O2 for them? What is their baseline PaO2 value? Many patients with chronic respiratory disease live quite happily with a PaO2 of 7.5 kPa. Check the results of previous ABGs when the patient was well to see how much they have decompensated. Next check if the patient is retaining CO2. This will determine the amount of O2 therapy you can give and will guide you to the degree that a respiratory component is determining their blood gas. The body never overcompensates; so if the PaCO2 is high, the patient is not ventilating adequately. The pH will tell you if the acidosis is of respiratory origin or if it is compensatory (if there is a metabolic alkalosis and the patient is hypoventilating to try and compensate for this). Remember that the O2 saturation curve for haemoglobin (Hb) is a steep sigmoid curve that starts to plateau around a PaO2 of 9.1 kPa (70 mmHg). This means a patient may have a PaO2 of 8.5 kPa and still have better than 90% O2 saturation. However, in the steep part of the curve (PaO2 of 6–8 kPa), small changes in PaO2 dramatically affect O2 saturation and are clinically significant. Check to see if the pH is normal or if there is an acidosis (pH <7.35) or an alkalosis (pH >7.45). If either exists, decide if it is metabolic or respiratory and whether or not it is compensated (see Table 13.2). Table 13.2 Interpreting acid–base disorders. Reproduced from Zilva J.F., Pannall P.R., Mayne P. (1989) Clinical Chemistry in Diagnosis and Treatment. Lloyd-Luke (Medical Books) Ltd, London. A compensated acid–base disorder suggests chronic disease, over days to weeks, whilst an uncompensated acidosis or alkalosis suggests a more acute problem. In general, the body compensates for changes in pH by altering the respiratory rate (e.g. in metabolic acidosis, CO2 is ‘blown off’) and regulating renal excretion of HCO3. Respiratory compensation occurs much quicker (minutes) than renal compensation (hours to days). If the patient is acidotic, you will need to calculate the anion gap ([Na + K] − [Cl + HCO3]) in order to refine the differential diagnosis. A raised anion gap is due to the addition of unmeasured acid to the system, for example, methanol, urea, diabetes (ketones), paraldehyde, isoniazid, lactic acid (in anaerobic metabolism), ethanol and salicylic acid. A good mnemonic for this is ‘MUD PILES’. A normal anion gap acidosis is usually due to loss of base, for example, from use of acetazolamide or other carbonic anhydrase inhibitors, diarrhoea and renal tubular acidosis. If stable, their ABGs will reveal a compensated respiratory alkalosis. If stable, their ABGs will reveal a compensated respiratory acidosis. They will often have a very high bicarbonate. Patients with type 1 failure progress to type 2 as they tire. Female nurses usually (but not always) catheterize women; you will certainly end up catheterizing men (whether you are a man or a woman). Suprapubic catheterization is a useful last resort. It is useful to perform a bladder scan to get an idea of the bladder volume before such a procedure. Same equipment as for the men Be aware of underlying causes for problems in catheterization such as tumours. If concerned, do a PV and call for help. If you are going to the trouble of doing blood cultures, it is better to do two sets from different sites, particularly if accurate diagnosis is important. Three sets for infective endocarditis are the gold standard. Vacutainers are much quicker to use than needles and syringes but have no ‘flashback’ to let you know when you have entered the vein. Ask the patient for their preferred arm. Obvious forearm or cubital fossa veins are good. In dialysis patients, never use their arteriovenous (AV) fistula arm. Likewise, never use an arm with lymphoedema or one that has had its lymph nodes removed or irradiated. (See Fig. 13.3) Cannulation often worries junior doctors as it is something that they are routinely required to do and is initially a difficult skill to acquire. Anyone who tells you that they never had trouble cannulating is lying! That said, it is a skill that anyone can acquire but practice really is the only way to be good at this. Here we outline the basic procedure but the best way to learn is to get someone with good technique to show you. Table 13.3 Cannula sizes.
PRACTICAL PROCEDURES
General hints
Arteries in order of preference
Have ready
The procedure
If you fail
Hints
Interpreting ABGs
pH
7.35–7.45
PCO2
4.3–6.0 kPa
Base excess
±2 mmol/l
PO2
10.5–14.0 kPa
Serum HCO3−
22–26 mmol/l
O2 saturation
95–100%
Points to consider when interpreting ABGs
PO2 and PaCO2
pH
pH
PCO2
HCO3−
K+
Acidosis
Metabolic – early
↓
Normal
↓
Usually ↑
Metabolic – compensated
Normal
↓
↓
Normal or ↓
Respiratory – early
↓
↑
Normal or ↑
↑
Respiratory – compensated
Normal
↑
↑↑
Alkalosis
Metabolic – early
↑
Normal
↑
↓
Metabolic – compensated
↑
Normal
↑↑
↑
Respiratory – early
↑
↓
Normal or ↓
↓
Respiratory – compensated
Normal
↓
↓↓
Normal or ↑
Serum electrolytes (Na+, Cl− and HCO3)
Hints
Respiratory disease and ABGs interpretation
Type 1 respiratory failure:
Type 2 respiratory failure:
Bladder catheterization
Men
Have ready
The procedure
If you fail
Women
Have ready
The procedure
Blood cultures
Have ready
The procedure
Hints
Venepuncture
Have ready
The procedure
Choosing a vein
If you can’t find a vein
Hints
Cannulation (Venflon/line insertion)
Have ready
Colour
Size
Use
Yellow
24G
Paediatric or oncology patients and intravenous (IV) drug users
Blue
22G
Small, fragile veins
Pink
20G
Regular IV drugs and fluid administration
Green
18G
Blood transfusions and fluids
White
17G
As above – rarely available
Grey
16G
Rapid fluid administration, GI bleeds
Brown/orange
14G
Major bleeds, usually placed prophylactically in theatre
The basic procedure