P
pacemaker see artificial pacemaker
pacinian corpuscles specialized nerve endings subserving vibrational awareness, located deep within dermis; formed as lamellated bulbs at termini of A-beta sensory nerve fibres (see Table S2)
packed cell volume (postcentrifuge) volume of blood cells in a sample of blood
packing infilling a wound with dressing material
pad see individually named pads; Table P1 and Figure P1
Pad type | Examples | Description |
---|---|---|
Digital pads | Plantar bar/long prop | SCF pad formed to infill the plantar aspect of the shanks of lesser toes, in order to prevent/reduce overcontraction of one or more lesser toes |
Dorsal bar | SCF pad formed to infill the dorsal aspects of one or more hammered or retracted lesser toes, to reduce trauma to the skin overlying the prominent interphalangeal joints | |
Dorsoplantar splint | SCF pad made as a combination of the plantar and dorsal bars, to correct digital deformity/reduce trauma to the apices and dorsa of lesser toes | |
Interdigital wedge | SCF or foam (plain, cavitied or holed) pad formed to match the dimensions of the interdigital space to reduce reformation of an interdigital heloma molle | |
Dorsal proximal/distal/apical/interdigital crescent | A crescent-shaped pad applied proximal/distal to the dorsal/apical/interdigital area of a hyperkeratotic lesion on a digit, to reduce local pressure and friction | |
Dorsal horseshoe | A horseshoe-shaped pad, where the ‘arms’ of the horseshoe cover the dorsal aspects of toes adjacent to the digit affected by a corn, and the U acts in the same manner as a crescent pad to protect the lesion | |
Plantar metatarsal pads | Plantar cover | A pad that covers the plantar skin of the forefoot, from the webbing to a line approximately 1 cm distal to the bases of the metatarsals |
U’d plantar cover | A plantar cover into which a U has been cut to deflect pressure away from a plantar lesion. The U may be infilled with cushioning material | |
Winged plantar cover | A plantar cover into which semicircular cutouts have been made, to deflect pressure from the 1 and/or 5 MTPJs | |
Plantar metatarsal pad | A pad applied to the 2/3/4 metatarsals, the distal limit of which applies pressure to the 2/3/4 metatarsal heads so that the 2/3/4 MTPJs are extended and the 2/3/4 toes realigned into a more functional position; the pad will also reduce compression between adjacent metatarsal heads | |
Plantar bar | A pad similar to a plantar cover, the distal limit of which had been shaped to accommodate up to 5 U’d areas | |
Shaft pad/long shaft pad | A pad applied to an individual metatarsal to allow sagittal-plane realignment | |
Others | D filler Valgus pad | A pad that is shaped to infill the plantar aspect of the medial longitudinal arch to reduce excessive pronation or ease the pain of foot strain |
Hallux valgus oval | An oval pad, with or without a central cavity or hole, that is applied to the medial aspect of the 1 MTPJ to reduce local shear stresses in cases of HAV | |
Heel pad | A pad shaped to the plantar aspect of the heel, to cushion or reduce pressure to a plantar bursitis or heel spur | |
Posterior heel pad | A pad designed to deflect pressure from the posterior lateral area of the heel, in cases with Haglund’s deformity | |
Doughnut pad Ring pad Oval pad | A circular pad with a central cavity or hole applied to the plantar aspect of the heel to protect the point of insertion of the plantar fascia | |
Cobra pad | A pad that combines a medial heel wedge, a valgus filler and a medial forefoot pad, to reduce excess foot pronation | |
Dumbbell pad | A pad that combines the action of a shaft pad to dorsiflex an individual metatarsal head, and an interdigital wedge, to reduce friction and pressure at the depth of the interdigital sulcus | |
Achilles tendon pad | A pad applied to the posterior aspect of the heel, to reduce pressure and friction at the insertion of the tendo Achilles |
SCF, semicompressed felt; MTPJ, metatarsophalangeal joint; HAV, hallux abductovalgus.
padding materials clinical materials used specifically to cushion, deflect pressure from or protect an area, substitute for lost tissue; adhesive padding materials include felts, polyurethane and polyethylene sheet foam and fleecy web that are shaped and stuck directly to skin, added to insoles or added to the shoe inner (see individually named pads Tables P1 and P2 and Box P1)
Material | Function |
---|---|
Compressed felt | To apply moderate leverage to a segment of a foot, such as a forefoot or rearfoot wedge |
Semicompressed felt | To protect vulnerable tissues and reduce the rate of reformation of hyperkeratotic lesions, such as after the enucleation of corns, by cutting a U, a wing, a cavity or a hole in the padding material to correspond to the site of the lesion To reduce compression on soft tissues imposed e.g. by ground reaction forces and underlying bony prominences To substitute for lost or atrophic tissues, e.g. overlying a bony prominence To carry a medicament, e.g. where salicylic acid paste is applied to a lesion by locating the medicament in a cavity within the semicompressed felt pad |
Felt and foam | To combine the benefits of both semicompressed felt and polyethylene foam in one material |
Polyurethane foams | To cushion an area lightly To infill a cavity, wing or U in a semicompressed felt pad |
Polyethylene foams | To provide a degree of cushioning, e.g. where soft tissues have undergone atrophy To infill a cavity, wing or U in a semicompressed felt pad |
Fleece/fleecy web | To aid retention of a small sterile gauze dressing To reduce friction at the skin surface, after local callosity has been debrided To reduce shear stress in areas of tissue subject to bursa formation |
Box P1 Principles of clinical digital padding
Paget’s disease see disease, Paget’s
pain control therapeutic measures used to reduce or resolve all components of pain, including analgesic drugs (e.g. paracetamol, non-steroidal anti-inflammatory drugs [NSAIDs], disease-modifying antirheumatic drugs [DMARDs], opioids and anxiolytics), electrical stimulation of the inhibitory control mechanisms (e.g. transcutaneous electrical nerve stimulation [TENS]), hypnosis and other cognitive and behavioural approaches (Table P3)
pain scale an objective value of subjective pain; value (between 0 and 10) is ascribed by the patient to the pain experience, where 0 = no pain at all, and 10 = worst pain experienced/imaginable; used to note pain in relation to daily activities, or document pain changes over a period of time (e.g. a week or month) in relation to a specific therapeutic interventions
Figure P1a Pads. A, combined dorsoplantar splint. B, adapted combined dorsoplantar splint to obtain deflection from dorsal and apical lesions. C, bolster pad for digits 2–4 when correction cannot be achieved. The bolster deflects pressure away from the apices. This article was published in Neale’s Disorders of the Foot, Lorimer, French, O’Donnell, Burrow, Wall, Copyright Elsevier, (2006).
Figure P1d Strapping for plantar metatarsal padding. This article was published in Neale’s Disorders of the Foot, Lorimer, French, O’Donnell, Burrow, Wall, Copyright Elsevier, (2006).
pain sensation evaluation see Table M13
painful heel see heel pain; Table H9
painful neuropathy presentation of sensory neuropathy predominantly affecting patients with diabetes, affecting one or both feet or legs, in a general or patchy distribution, variably as constant burning sensations, paraesthesiae, ‘crawling’ or shooting pains, or exquisite contact discomfort; affected patients may lose weight, and develop motor neuropathy (proximal diabetic myotrophy); persists for ~ 6–18 months, and heralds profound sensory loss; may respond to mild antidepressant agents (e.g. amitriptyline and diazepam) and anticonvulsant drugs (e.g. gabapentin), but is resistant to normal analgesics
palindromic relapsing; recurring
palliation; palliative therapies that reduce symptom severity, but do not cure
palpation examination using the hands, e.g. to outline underlying muscular, vascular or neural structures, determine joint mobility and tissue resistance palpate masses
panarthritis inflammation affecting all joints
pancytopenia marked reduction in numbers of circulating blood cells
panniculitis inflammation of the forefoot plantar fibrofat pad
papilloma benign epithelial neoplasm
papule small, solid, circumscribed, elevated skin lesion
paracetamol see acetaminophen; Table P3
paraesthesia abnormal sensation, e.g. burning, tingling, pricking; i.e. ‘pins and needles’
paraffin gauze; tulle gras low-adherence gauze or mesh dressing, impregnated with paraffin wax; may also incorporate an antiseptic (e.g. chlorhexidine gluconate) (see Table D10)
paraffin wax baths see wax baths
parallel pitch lines parallel lines drawn on a lateral radiograph of rearfoot, to determine the degree of prominence of posterosuperior eminence of calcaneum (see Table R3; Figure P2)
paraparesis mild paralysis of legs and feet
paraplegia paralysis of lower trunk and both legs
parasympathetic relating to the parasympathetic division of the autonomic nervous system
parasympathomimetic an agent whose action causes effects resembling parasympathetic activity
parathyroid gland see gland, parathyroid
parenchyma cells characteristic of an organ, contained within and supported by the stroma
parenteral administration of a medicinal or therapeutic substance, other than through the gastrointestinal or respiratory tracts, e.g. by intravenous, intramuscular or subcuticular injection
Figure P2b Parallel pitch lines (PPLs) to determine the prominence of the bursal projection (BP or posterosuperior prominence). PPL1 is the baseline, tangential line to A anterior tubercle, and M medial tubercle of posterior tuberosity; the perpendicular d is drawn between between PPL1 and T posterior lip of talar articular facet. The bursal projection BP touching or below PPL2 is normal, not prominent. This article was published in Neale’s Disorders of the Foot, Lorimer, French, O’Donnell, Burrow, Wall, Copyright Elsevier, (2006).
paresis partial or complete paralysis
Paris point unit of the continental shoe sizing system; there is 6.66 mm between each Paris point (see Table S4)
parkinsonism; Parkinson’s disease; paralysis agitans; shaking palsy see disease, Parkinson’s
paronychia nail fold/sulcus inflammation and infection, often associated with ingrowing toenail
paroxysm spasm of severe pain, of sudden onset
partial dislocation see subluxation
partial nail avulsion see nail avulsion, partial
partial rupture of Achilles tendon see Achilles tendinitis; Achilles tendon rupture
partial-thickness burns destruction of epidermis and superficial dermis, by dry heat
patch test; patch testing see test, patch
patella; kneecap large sesamoid embedded within quadriceps tendon at anterior aspect of knee joint; its cartilaginous internal facet articulates with knee joint; exerts pulley-like action within the patellar groove; acts as a fulcrum for quadriceps action, centralizing action of contributory muscles (i.e. rectus femoris, vastus intermedius, vastus lateralis and vastus medialis) (see Table J1); protects quadriceps tendon, allowing knee extensor apparatus to take high load (by lengthening the lever arm, and giving mechanical advantage to quadriceps)
patellar groove anterior groove (between medial and lateral femoral condyles) for patellar travel, under quadriceps correct patellar tracking is maintained by patellotibial and patellofemoral ligaments; misalignment of the patella within patella groove causes anterior knee pain (see syndrome, runner’s knee)
patellar tendon reflex see reflex, patellar
patellofemoral joint syndrome see syndrome, runner’s knee
pathogenesis mode of origin or development of any disease or morbid process
pathognomonic characteristic symptoms of a disease
pathological callus hyperkeratosis formed in response to local stresses or biomechanical dysfunction, and of significance to patient/clinician (see Table C15)
pathology tests analysis of tissue specimens, e.g. to identify tissue (histopathology), chemical constituents of body fluids (chemical pathology) or confirm type/sensitivity of any infecting organisms (microbiology), in order to indicate the most effective patient management regime; see sampling
pathophysiological pain; second pain; slow pain pain that encourages healing by inducing protective behaviours; it originates from stimulation of high-threshold polymodal nociceptors (free nerve endings, present in all tissues and responsive to mechanical, chemical and thermal stimuli) and is transmitted along slow-conduction C fibres (which also induce emotional and behavioural responses to pain via thalamic connections, and activate inhibitory pain pathways and the release of endogenous opioids)
pauciarticular affecting only one or two joints
paucibacilliary leprosy tuberculoid leprosy; TT leprosy; see Table H7
PEDIS ulcer classification system see Table U2
pedobaroscope calibrated plantar pressure visualization device, Table G2
pellagra niacin (nicotinic acid) deficiency (e.g. in chronic alcoholism)
‘pencil in cup’ deformity erosive inflammation characteristic of metatarsophalangeal joints in rheumatoid or severe psoriatic arthritis (arthritis mutilans); metatarsal head is eroded and the base of the adjacent proximal phalanx becomes increasingly concave see metatarsal head dystrophy
penicillin antibiotic derived from Penicillium moulds; bactericidal against non-resistant Gram-positive microorganisms; may provoke sensitivity reactions, e.g. urticaria and/or anaphylaxis; note: patients with penicillin allergy may also react to penicillamine and cephalosporins
penicillinase enzyme inactivating penicillin; produced by some staphylococci
pencilling of metatarsals see metatarsal head dystrophy
percentage mass calculation to convert drug concentration in solution (%) to mass (in mg); based on the principle that 1% solution contains 10 mg of drug per 1 mL of solution (see Table M5)
percutaneous passage of substances through unbroken skin
perforated film absorbent dressings primary wound dressing; see Table D10
perfusion passage of blood and tissue fluid through the capillary bed
periarteritis inflammation of the tunica adventitia of an artery
periarthritis inflammation of tissues surrounding a joint
pericarditis inflammation of pericardial membrane, e.g. in autoimmune disease
pericardium fibroserous, double-layered membrane surrounding the heart
periostitis inflammation of periosteum
peripheral tissues/structures furthest from the centre
peripheral arterial disease; PAD see disease, peripheral vascular (Table P4)
Test modality | Diagnostic indicators |
---|---|
Medical history | • Evidence of atherosclerosis, such as ischaemic heart disease, cerebrovascular disease • Risk of atherosclerosis, such as cigarette smoking, diabetes mellitus |
Pulses in the lower limb | Popliteal, posterior tibial, dorsalis pedis and peroneal pulses • Pulse quality (bounding, full, normal, weak, absent) • Rate (beats/minute) • Regularity (regular, irregular, regularly irregular, irregularly irregular) |
Presenting symptoms | • Cold, numb feet • Intermittent claudication • Rest pain • Painful ulceration |
Ankle–brachial index (ABI) | • 0.9–1.1 = normal • 0.7–0.9 = PAD, some compromise of tissue viability • 0.5–0.7 = severe PAD, compromised tissue viability • <0.5 = threat of ischaemic gangrene/very poor tissue viability • >1.2 = calcification of tunica media of leg/foot artery, compromised tissue viability |
Segmental systolic pressure | Serial measurement of systolic blood pressure along length of limb: • A sudden decrease indicates the location of the vascular obstruction |
Venous filling time Note: this test is not valid if the patient has venous incompetence | The time it takes for veins to refill in a limb that has been drained of venous blood (with the patient supine, the leg is elevated to 45 ° for 1 minute, then the leg placed in a dependent position and the time taken for the dorsal foot veins to refill) • <15 seconds = normal • 20–30 seconds = moderate ischaemia • >40 seconds = severe ischaemia |
Buerger’s test | The observation of the change in skin colour in response to limb elevation and dependency (with the patient supine, the leg is elevated to 45 ° for 1 minute, then the leg is placed in a dependent position and the time taken for normal skin colour to return is noted) • <10 seconds = normal response • Patchy persistent rubor indicates limb ischaemia • Persistent pallor (>10 seconds) indicates limb ischaemia • Persistent cyanosis indicates limb ischaemia |
Buerger’s angle | The colour response of the sole of the foot to limb elevation (with the patient supine, the leg is elevated and the angle of limb elevation is noted when the skin of medial longitudinal arch shows pallor) • 60–70 ° = normal arterial supply to foot • < 45 ° = compromised arterial supply to foot • < 30 ° = severe compromised arterial supply to foot |
Capillary refill time Note: this test is not valid if the patient has signs of current Raynaud’s disease | The time taken for normal skin tone of a horizontal limb to return after the nail bed or digital pulp has been compressed by thumb pressure • <5 seconds = normal response • >5 seconds = some ischaemia • >15 seconds = marked ischaemia |
Doppler sounds | • Triphasic = normal • Biphasic = normal/some loss of arterial elasticity • Monophasic = loss of arterial elasticity/arterial stenosis • Loud = high rate of blood flow • Quiet = slow rate of blood flow • No sound = no blood flow (proximal arterial occlusion) |
Skin temperature Note: this test is not valid if the patient has signs of current Raynaud’s disease | • ~ 31 °C = normal foot skin temperature • <29 °C = possible poor skin perfusion |
peripheral pain receptors see nociceptors
peripheral pulses see pulse, dorsalis pedis
peripheral vascular disease generic term denoting reduced arterial supply to, and compromised venous/lymphatic return from, the lower limbs and feet; see disease, peripheral vascular
peristalsis waves of alternate contraction and relaxation in circumferential muscle tissue of a tubular structure, driving contents forward, e.g. movement of blood through the vascular system (see law, Starling’s)
peritendinitis inflammation of Achilles peritendon, due to e.g. acute local trauma, chronic overuse, infection or musculoskeletal disease
periungual fibroma see Koenen’s tumour
permeability passage of substances through a membrane
permethrin; Lyclear topical scabies treatment
peroneal cuboid see syndrome, peroneal cuboid
peroneal muscular atrophy see disease, CMT disease type I
peroneal pulse see pulse, dorsalis pedis; peroneal artery
peroneal spastic flat foot see tarsal coalitions
peroneus; peronei peroneal muscles (longus [PL] and brevis [PB]) of lateral compartment of lower limb; action eversion of subtalar joint and plantarflexion of foot at ankle joint; PL and PB tendons lie within a common synovial sheath, within the retrofibular groove at the posterior aspect of the lateral malleolus, and enter the foot deep to the superior peroneal retinaculum (creating a point of ischaemic stress, leading to chronic degeneration and degenerative changes in PL and PB tendons; see subluxing peroneal tendons); PL and PB common tendon sheath divides to form individual sheaths at lateral side of foot
peroneus brevis; PB extrinsic foot muscle within the lateral compartment of lower leg
peroneus longus; PL extrinsic foot muscle, within lateral compartment of lower leg