Cardiovascular system



Cardiovascular system



The more common cardiovascular presenting problems are chest pain, hypertension, edema, murmur, mitral valve prolapse, palpitations, congestive heart failure, acute ischemia, abnormal stress tests, arrhythmias, congenital heart disease, syncope (fainting), hyperlipidemia, and claudication.


A cardiologist is a physician who specializes in diseases of the heart and vessels. A cardiothoracic surgeon is a physician who specializes in surgical procedures of the heart and chest. A variety of physicians frequently use the codes, as you will see as you code the services and procedures within this chapter. Examples of procedures include valve repair, beating heart surgery, aortic dissections, and excision of tumors of the chest wall.


When coding cardiovascular services, the coder will commonly use codes from the Evaluation/Management, Surgery, Medicine, and Radiology sections of the CPT manual.



Evaluation and management services


Often the E/M services provided to a patient with a cardiac condition are very complex and extensive. The history and physical examination of the patient with a suspected cardiovascular condition are of critical importance to proper medical management of the patient. The physician has training, skills, knowledge, and experience that cannot be replaced by a laboratory test; rather, the tests assist the physician in the diagnosis process. Through the history and physical examination, the physician gathers a wide range of information necessary to diagnose the patient. For example, the symptom of chest pain, which is a cardinal manifestation of cardiac disease, could be caused by conditions of the aorta, pulmonary artery, bronchopulmonary tree, pleura, mediastinum, esophagus, diaphragm, tissues of the neck or thoracic wall (including the skin, thoracic muscles, cervicodorsal spine, costochondral junctions, breasts, sensory nerves, or spinal cord), stomach, duodenum, pancreas, or gallbladder. There are many potential causes for just one of the symptoms of cardiac disease.


Consultations are a frequent cardiologist service. Cardiology consultations can be provided to the inpatient or outpatient, and the choice of the correct E/M code for the consultation is based on documentation of key components and contributing factors. The cardiology consultation often produces lengthy, complex medical reports.



Arteriosclerosis


Arteriosclerosis is a chronic disease of the arterial system that results in thickened and hardened walls of the vessels resulting in loss of the artery elasticity. Gradually, the arterial lumen narrows. This results in increased blood pressure because the heart has to pump harder to force the blood through the artery, weakening of the arterial walls that then become more susceptible to rupture, and insufficient perfusion to the tissues of the body. Atherosclerosis is a form of arteriosclerosis in which deposits of fat and fibrin (insoluble protein) accumulate on the vessel walls. These accumulations are atheromas. When referencing the Index under the main term “Atherosclerosis,” the coder is directed to “see Arteriosclerosis.” Under the main term “Arteriosclerosis, arteriosclerotic” the coder is directed to I70.90/440.9. In the Tabular, the code description indicates “Atherosclerosis” that is not further specified. There are more specific diagnosis codes available for assignment based on the location of the disease, such as aorta (I70.0/440.0), renal artery (I70.1/440.1), extremities (I70.209/440.2–), bypass graft of extremities (I70.309/440.3–), of other specified arteries (I70.8/440.8), or generalized or unspecified atherosclerosis (I70.91/440.9).


One commonly diagnosed condition is atherosclerosis of the coronary artery. To assign a code to this condition, you start by referencing “Arteriosclerosis, coronary” in the Index. You are then directed to I25.10/414.00. The code includes arteriosclerotic heart disease (ASHD), atherosclerotic heart disease, and coronary artery sclerosis.


ICD-10-CM: The assignment of additional characters is based on the location of the atherosclerosis and other factors, such as with or without angina pectoris (heart pain). For example, the following more common diagnoses are reported with six-character codes:



ICD-9-CM: The fifth-digit code classifications are also based on the location of the atherosclerosis. For example, the following fifth-digit classifications:



Review the subterms listed under the main term “Arteriosclerosis, arteriosclerotic” in the Index to become familiar with the entries located there and then review the codes you are directed to from the Index.



CASE 6-1   6-1A Cardiothoracic Surgery Consultation


CASE 6-1


Time to put your cardiology coding knowledge to work by coding an E/M service provided by a cardiologist. A decision to perform surgery was made during this E/M visit, so remember to use the modifier that will indicate it.



6-1A  Cardiothoracic surgery consultation


LOCATION: Inpatient, Hospital


PATIENT: Manuel Lopez


PHYSICIAN: David Barton, MD


REASON FOR CONSULTATION: Atherosclerotic heart disease


HISTORY OF PRESENT ILLNESS: This 62-year-old Hispanic male was being considered for knee replacement and in his preoperative workup underwent a stress test, which did not show any ischemia; however, because of angioplasty 6 months ago, the patient was considered a candidate for angiography.


CARDIAC RISK FACTORS: Risk factors include a remote history of cigarette smoking, hypertension, previous coronary stent, dyslipidemia, and adult-onset diabetes mellitus.


MEDICAL HISTORY: Previous operations include the following:



CURRENT MEDICATIONS:



FAMILY HISTORY: Positive for coronary disease


SOCIAL HISTORY: The patient is married and lives with his wife in Manytown. He drinks minimally and stopped smoking years ago.


REVIEW OF SYSTEMS: Review of systems is significant for bilateral knee osteoarthritis. Echocardiogram done last year showed normal ventricular size with concentric hypertrophy and apical area of aneurysm.


PHYSICAL EXAMINATION: On examination, the patient is a 272-pound, 6-foot Hispanic male in no apparent distress, supine after his cardiac catheterization. Jugular venous pressure is normal. Carotids were 2+, equal, and quiet. The CHEST is clear and equal. The HEART has a regular rhythm with a rate of 70 without murmur, gallop, or rubs. The ABDOMEN is soft and obese without organomegaly. The upper and lower EXTREMITIES show no cyanosis, clubbing, or edema. Pulses are intact peripherally. The patient is grossly and neurologically intact. The chest x-ray shows normal cardiothoracic ratio. LUNG fields are clear.


The ECG (electrocardiogram) shows normal sinus rhythm with anterolateral ST (sinus tachycardia) segment and T-wave changes.


LABORATORY STUDIES: The laboratory shows sodium of 135, BUN (blood urea nitrogen) of 24, glucose of 115. The lipids are within satisfactory limits. The protime is 12.7 and PTT (partial thromboplastin time) is 31.6. White blood cell count is low at 3.5. Hemoglobin is 13.6, and platelets are 176,000.


Cardiac catheterization by Dr. Elhart 6 months ago showed good left ventricular contractility with, perhaps, some anterior early relaxation. The dominant right coronary artery had a 70% lesion at the posterior descending proximally. The left main was narrowed 40% distally. The left-to-right and right-to-left fill. The diagonal branch was diseased but small. The left circumflex was narrowed at two obtuse marginal branches at 70% to 90% proximally, respectively.


IMPRESSION: Three-vessel atherosclerotic heart disease in a 62-year-old Hispanic male with adult-onset diabetes mellitus, normal left ventricular function, and need for knee replacement.


DISPOSITION: The patient will be retained in the hospital and will undergo coronary vascularization tomorrow. Operation, complications including blood transfusion, risks, and alternatives were discussed with the patient and his family.



You will be coding other cardiovascular E/M services throughout this chapter.



Coronary artery bypass grafts


The Surgery section, Cardiovascular System subsection, codes are divided into Heart/Pericardium (33010-33999) and Arteries/Veins (34001-37799). In the Arteries and Veins subheading, you will find many of the same types of procedures that are found in the Heart and Pericardium subheading, except the Arteries and Veins are for procedures on noncoronary vessels elsewhere in the body.


When coronary arteries clog with plaque (arteriosclerotic coronary artery disease [ASCAD]), the flow of blood is lessened. Figure 6-1 is a drawing that was placed in the patient’s medical record by the cardiologist to indicate the blockage of the patient’s heart vessels. Note that the figure indicates the percentage of blockage of the involved vessels. For example, the right coronary artery (RCA) is 100% blocked by plaque. Figure 6-2 illustrates an artery on fluoroscopy that is blocked with plaque. The heart muscle may begin to function below normal levels—reversible ischemia. If the heart muscle is denied adequate blood flow for an extended period, the muscle may die—irreversible ischemia.




A coronary artery bypass graft (CABG) bypasses the clogged area(s) of the vessels to improve blood flow. There are three types of coronary artery bypass grafts:



The heart has two main coronary arteries—the left (left main) and right (right coronary artery). The left main (LM) artery divides into the left anterior descending (LAD) and left circumflex (LCA) arteries (Figure 6-3). The right coronary artery (RCA) supplies the right ventricle and continues down the back of the heart (posterior aspect), where it is called the posterior descending artery (PDA). The coronary artery can be bypassed with an artery using the internal mammary artery, gastroepiploic artery, epigastric artery, radial artery, and arterial grafts from other areas. Note in Figure 6-3 that the internal mammary artery (originates from the subclavian artery) is left attached to the subclavian artery on one end, and the distal end is detached from its origin and reattached to the coronary artery to bypass the area of damage. Other times, the surgeon removes a portion of a vein and uses it for a graft, such as a right or left saphenous vein that is removed and used for a bypass graft. The procurement of the artery or vein is included in the CABG code and would not be reported separately. If an upper-extremity artery, such as the radial artery, is harvested for grafting, however, the harvesting service can be reported separately with 35600 (Harvest of upper extremity artery, one segment for coronary artery bypass procedure) or harvesting of an upper-extremity vein can be reported separately with add-on code 35500 (Harvest of upper-extremity vein, one segment for lower extremity or coronary artery bypass procedure).



Codes are selected based on the type (venous or arterial) of graft (harvested) and the number of grafts placed on the coronary artery (recipient). The number of grafts is determined by counting the number of distal anastomoses where the bypass graft is sutured to the diseased artery. For example, RCA and LAD receive two saphenous vein grafts. The type of graft is venous (harvested), and there were two grafts: one placed on the right coronary artery and one placed on the left anterior descending artery (recipients). The physician will often refer to the harvested and recipient, so you will need to know these terms to interpret the service provided correctly.


A CABG with venous and arterial grafts (33517-33530) is never used alone but only with the arterial graft codes (33533-33536). You can use the arterial graft codes alone (33533-33545) and the venous codes alone (33510-33516), but you can never report the Combined Arterial-Venous Grafting for Coronary Bypass codes alone. A helpful hint is to write “combination AV only” next to the codes 33517-33530 in your CPT manual as a reminder that these codes are used to report the venous graft(s) only when a combined arterial-venous graft is performed. Note that the codes from range 33517-33530 have a plus sign in front of them that indicates add-on codes and are never reported alone.


Cardiovascular surgeons often use artificial materials, such as Gore-Tex, to repair damaged areas of vessels. This artificial material is less susceptible to rejection and calcification than human tissue and can be readily available. The surgeon uses this material to repair a hole in a vessel as a seamstress would apply a patch to a hole in a pair of jeans. The material is cut to size and sewn over the hole. These artificial materials can also be formed into a tube and inserted into a vessel as a support for weakened or collapsed vessel walls.



CASE 6-2   6-2A Coronary Artery Bypass


CASE 6-2


Code the following CABG. Be certain to identify the number, location, and type of grafts.



6-2A  Coronary artery bypass


LOCATION: Inpatient, Hospital


PATIENT: Manuel Lopez


SURGEON: David Barton, MD


PREOPERATIVE DIAGNOSIS: Atherosclerotic heart disease


POSTOPERATIVE DIAGNOSIS: Same


PROCEDURE PERFORMED: Coronary artery bypass grafts × 4 with left internal mammary artery to left anterior descending bypass and sequential saphenous vein bypass from the aorta to the first and second obtuse marginal branch of the left circumflex with an ongoing graft to the posterior descending coronary artery.


ANESTHESIA: General


INDICATIONS: This 62-year-old Hispanic male with a history of degenerative knee disease was considered a candidate for orthopedic surgical management; however, preoperatively he underwent stress testing, which was equivocal but prompted angiography, which showed severe three-vessel disease with normal ventricular function.


FINDINGS AT SURGERY: The vein was a 4-mm (millimeter)-diameter vessel of good quality and was used in reverse fashion. The left internal mammary artery was a 1.5-mm-diameter vessel of good quality. The left anterior descending was a 2-mm-diameter vessel of good quality. The first and second obtuse marginal branches were both 2 mm in diameter and of good quality. The posterior descending was, likewise, 2 mm in diameter and of good quality. All the grafts were appropriate prior to closure and were placed distal to palpable disease.


DESCRIPTION OF PROCEDURE: The patient was brought to the operating room and placed in the supine position. With the patient under general intubation anesthesia, the anterior chest, abdomen, and legs were prepped and draped in the usual manner. A segment of the greater saphenous vein was harvested from the left thigh using the endoscopic vein-harvesting technique and prepared for grafting. The pericardium was incised sharply and a pericardial well created. The patient was systemically heparinized and placed on single right atrial-to-aortic cardiopulmonary bypass with a stump in the main pulmonary artery for cardiac decompression. The patient was cooled to 26° C and, on fibrillation, aortic cross-clamp was applied and potassium-rich cold crystalline cardioplegic solution was administered through the aortic root with satisfactory cardiac arrest. Subsequent doses were given down the vein graft as the anastomosis was completed and also via the coronary sinus in a retrograde fashion. The end of the greater saphenous vein was then anastomosed to the proximal third of the posterior descending coronary artery using 7-0 Prolene. The graft was brought to the patient’s left and then anastomosed side to side to the second obtuse marginal branch, followed by the first obtuse marginal branch, all with 7-0 continuous Prolene. The left internal mammary artery was then brought down to the midportion of the left anterior descending and anastomosed thereto with 8-0 continuous Prolene. The aortic cross-clamp was removed after 62 minutes with spontaneous cardioversion to a normal sinus rhythm. The patient was then warmed to 37° C esophageal temperature, during which time the vein graft was trimmed to size and anastomosed to the ascending aorta using 5-0 continuous Prolene technique. The patient was weaned from cardiopulmonary bypass without difficulty using no inotropes after 99 minutes. The patient was decannulated, protamine was given, and hemostasis was obtained. Temporary pacer wires were placed from the right atrium and right ventricles. The chest was drained with two Argyle chest tubes and closed in layers in the usual fashion. The leg was closed similarly. Sterile compression dressings were applied. The patient returned to the surgical intensive care unit in satisfactory condition. Sponge and needle counts were correct × 2.





Pacemaker


A pacemaker is an electrical device that is inserted into the body to shock the heart electrically into regular rhythm. The two parts of a pacemaker are the battery and electrode. The electrode is the device that emits the electrical charge. The electrode is also called the lead and is a flexible, thin tube. The battery is also called a pulse generator. Some generators are programmable and have a wide range of programming options. The pulse generator is placed into a pocket either under the clavicle, as illustrated in Figure 6-4, or under the muscle of the abdomen below the rib cage.



Either an epicardial or transvenous approach can be used to implant the electrode portion of the pacemaker. The epicardial approach involves opening the chest to the view of the surgeon and the device being placed on the heart. The transvenous approach is most commonly used because it is the least traumatic to the patient and involves inserting a needle with a wire attached (guidewire) into a vein. The guidewire then directs the placement of the electrode into the heart while the surgeon views the progression using a fluoroscope. The electrode is then attached to the pulse generator.


The pacemaker can be a single- or dual-chamber unit. A single-chamber pacemaker uses one pulse generator and one electrode, which is placed in either the atrium or the ventricle. A dual-chamber pacemaker uses a pulse generator and two electrodes—one placed in the atrium and the other placed in the ventricle.


Pacemakers can be permanent or temporary. A temporary pacemaker can be used when the heart needs only short-term pacing support. For example, when a patient is waiting for placement of a permanent pacemaker or a patient is experiencing postsurgical cardiac instability. After the pacemaker is placed, the physician will test the device to ensure that it is operating correctly. The pacemaker implantation report will include a statement such as “thresholds were obtained and were adequate.” The testing and setting are included in the implantation service and are not reported separately. Special or extensive pacing, if noted in the report as being above the usual service, can be reported separately.


A cardioverter-defibrillator is an implantable electronic device that has both the pulse generator and the electrodes, but it is capable of more functions than the pacemaker. The device senses irregularities in the heart rhythm and emits electrical charges to regulate the heart rhythm and pace the heart to correct heart rhythm. The device can be programmed to sense a wide variety of heart irregularities and then further programmed to emit various electrical charges in response. The device is used for antitachycardia (stops rapid heartbeat) pacing, low-energy cardioversion (restoration of normal heartbeat), or defibrillating shocks to treat tachycardia or fibrillation (quivering).


A single-chamber cardioverter-defibrillator has a lead or leads inserted into a single chamber; a dual-chamber cardioverter-defibrillator has leads inserted into both the atrium and ventricle. The number of leads used does not indicate a single- or dual-chamber cardioverter-defibrillator because when a single chamber is being paced, multiple leads into that single chamber may be used. It is the number of chambers into which the leads are placed, not the number of leads used, that impacts code choice.


During insertion, electrophysiologic testing of a cardioverter-defibrillator and electrical analysis of a pacemaker would often be conducted as a part of the insertion or reinsertion procedure. This testing and analysis would be reported separately using Medicine section codes 93279-93285. Perioperative testing is also reported separately with Medicine section codes 93286 and 93287. Remember to use modifier -26 if you are reporting only the professional component (physician) of the electrical analysis.


When a pacemaker or cardioverter-defibrillator battery is changed, it is actually the pulse generator that is replaced. As a part of the usual 90-day surgical package that accompanies the implantation service, follow-up visits would be considered part of the global service by the third-party payer and therefore not reimbursed.


Removal of only the pulse generator without insertion of a new pulse generator is reported with 33233 or 33241. If the pulse generator is replaced without insertion or replacement of transvenous electrode(s), both the removal and insertion of another pulse generator is reported with only one code. 33227-33229 (permanent pacemaker) or 33262-33264 (cardioverter-defibrillator), depending on the lead system number already in place. Removal and replacement of a permanent pacemaker pulse generator and transvenous electrode(s) is reported with 33233 in addition to 33234 or 33235 and 33206-33208. Removal and replacement of a cardioverter-defibrillator pulse generator and transvenous electrode(s) is reported with 33241 in addition to 33243 or 33244 and 33249.



Sick sinus syndrome


Sick sinus syndrome is a group of abnormal heartbeats (arrhythmias). The exact cause of this syndrome is unknown, but it is thought to be caused by a malfunction of the sinus node, which is the heart’s natural pacemaker. It can result in many arrhythmias, including tachycardia (fast heart rate), bradycardia (slow heart rate), sinus arrest, sinus node exit block, and sinus bradycardia. These arrhythmias are not reported separately because they are symptoms of the syndrome. Rather, the syndrome is reported. You can locate direction to the code in the Index by referencing “Syndrome, sick, sinus.”



CASE 6-3   6-3A Cardiology Follow-Up Note


CASE 6-3


With the information you just learned about pacemakers, code the following E/M services to a patient who, after several tests, will have a pacemaker implanted.



6-3A  Cardiology follow-up note


LOCATION: Outpatient, Clinic


PATIENT: Herbert Gillford


PHYSICIAN: Marvin Elhart, MD


DIAGNOSES:



MEDICATIONS: Tylenol; Lasix 40 mg (milligram) as needed; Vasotec 5 mg once daily; Colace; aspirin: Ambien; and Arthrotec.


I evaluated this patient approximately 2 years ago. He was seen by Dr. Pleasant again last year on referral from Dr. Green because of episodes of falling down. The episodes are significant to the point that he had a hip fracture. Apparently, he was transferred to a nursing home in his hometown.


The history from the patient is almost useless because he does not recollect any symptoms and he says he feels fine and has no shortness of breath or falling down. The patient has been diagnosed with dementia previously. We tried to contact his relative, but we got the answering machine.


We reviewed the previous notations from Dr. Green and Dr. Pleasant and myself. Predominantly the problem is that the patient has had repeated falls and has been diagnosed with sinus bradycardia with first-degree AV (arteriovenous) block and bundle-branch block; a Holter monitor read by myself recently revealed predominantly atrial block.


EXAMINATION today reveals an elderly man in a wheelchair in no acute distress. His blood pressure was 124/68 with a heart rate of 60. Lungs did not reveal any rales. Cardiovascular examination reveals distant heart tones with normal S1 (first heart sound) and S2 (second heart sound) with a holosystolic murmur, grade 1/6, over the mitral area. Abdomen was obese. Extremities: No edema.


ECG (electrocardiogram) in the office reveals him to be in sinus bradycardia with a rate of 49 with first-degree AV block and left bundle-branch block. The Holter monitor revealed him to be predominantly in atrial fibrillation; his minimum rate was 49 and maximum 114 with an average of 74. At that time, the notation did show that he was taking Lanoxin, which apparently he is not taking at this time.


An echocardiogram also read by myself revealed the patient to have dilated cardiomyopathy with severe LV (left ventricle) dysfunction with mild aortic insufficiency and significant mitral insufficiency.


IMPRESSION AND RECOMMENDATIONS: Symptomatic sick sinus syndrome with evidence of paroxysmal atrial fibrillation. It is very difficult to assess this patient symptom-wise because he does not have a lot of insight into his symptoms. He has been diagnosed with dementia. It seems to me that there is enough evidence that this patient’s falling down episodes might be related to his rhythm because that is the most common finding. The only solution to this problem would be implantation of a dual-chamber pacemaker.


We attempted to contact his family to discuss this and to obtain a consent, but we did not get an answer. We will contact the family again and discuss with them the recommendations. If they consent to it and agree to proceed, we will bring him in for implantation of a dual-chamber pacemaker.




In reports 6-5B and 6-5C, you will be coding the pacemaker implantation for the patient in report 6-3A, but you must learn a couple more things before you code the implantation service. Let us begin with echocardiography services.



Echocardiography


Cardiomyopathy (CMP) is a disease of the heart muscle. The exact cause(s) of cardiomyopathy are not known, and the symptoms of the disease are similar to those displayed in patients with myocarditis (infections of the heart muscle caused by virus, bacteria, or parasites), toxic effects of various drugs, myocardial infarction, various cardiac disorders (pericarditis, congestive heart disease, or stenosis), and various other conditions. As a part of the diagnostic process, the physician may order an electrocardiogram, chest x-rays, Doppler and echocardiography, radionuclide studies, cardiac catheterization, or blood tests.


Echocardiography obtains ultrasonic signals from the heart and coronary arteries with a two-dimensional image and/or Doppler ultrasound. Transthoracic echocardiography is a noninvasive procedure in which a transducer (transmitter) is placed on the skin and sound waves go through the structure of the body and bounce off the heart. From the frequency with which the waves return from the internal structures and bounce back to the transducer, the physician can determine the position and motion of the heart walls and internal structures of the heart and neighboring tissue.


Transesophageal echocardiography (TEE) is a procedure in which the transesophageal echography probe transducer is placed into the mouth and advanced into the esophagus, allowing a posterior view of the heart, as illustrated in Figure 6-5. Varieties of TEEs are reported with codes 93312-93318. The codes are divided into those done for congenital cardiac conditions (93315-93317) and those done for noncongenital cardiac conditions (93312-93314). The stand-alone codes 93312 and 93315 are for the full service, and the indented codes are for components of the service. For example, 93313 is used to report the placement of the transesophageal probe only. Code 93318 is used to report a TEE for monitoring purposes. These monitoring functions are not limited to monitoring of the heart but also include monitoring of numerous organs, such as the lungs and mediastinum. These monitoring services are not the usual diagnostic service but instead are urgent services that are required for a critically ill or injured patient. (In Case 6-14A, you will be coding a TEE service prior to a cardioversion.)



Doppler echocardiography is a technique that records the flow of blood through the cardiovascular system by tracking the movement of the red blood cells by ultrasound. The pulsed-wave or continuous-wave Doppler produces a video recording or strip chart detailing the force with which the blood passes through the cardiovascular system and the direction of the blood. A color-flow Doppler is similar to the pulsed-wave or continuous Doppler, but in addition to the color flow is a two-dimensional color display. The color makes identification of blood flow easier. Code 93306 is a complete transthoracic echocardiography that includes M-mode, when performed, and spectral and color-flow Doppler. A limited transthoracic echocardiography, without spectral and color-flow Doppler, is reported with 93307. Note that you cannot report 93320 (complete Doppler), 93321 (limited Doppler), or color-flow velocity mapping (93325) with 93307.



Valve insufficiencies


Insufficiency of a heart valve means the valve does not close properly, which may lead to hypertrophy (enlargement) of the heart because blood will pool in the chambers. Rheumatic heart disease is a result of rheumatic fever, which is a febrile (fever) disease caused by a streptococcal infection and results in valvular deformities. The ICD-10-CM/ICD-9-CM divides codes for nonrheumatic valvular insufficiencies from valvular insufficiencies that are a result of (sequela) rheumatic heart disease. Category I34/424 is for nonrheumatic or other specified causes. This code is also assigned for unspecified causes when only one valve is involved. The documentation is often aortic stenosis (I35.0/424.1) of unknown cause. When there are multiple valves involved in the diagnosis or the documentation specifies rheumatic, refer to I05/394 to I08/396. For example, a commonly documented diagnosis is aortic and mitral valve regurgitation (not specified as rheumatic), which is reported with I08.0/396.3. When both mitral and aortic valves are involved, the ICD-10-CM/ICD-9-CM presumes an etiology (cause) of rheumatic heart disease, and the physician does not need to document rheumatic heart disease. Also, when the diagnosis involves the tricuspid valve, the etiology also is presumed to be rheumatic heart disease if multiple heart valves are involved. In ICD-9-CM insufficiency of the pulmonary valve (424.3) is not considered to be rheumatic when other heart valves are also involved unless stated as rheumatic (397.1).



CASE 6-4   6-4A Echocardiogram


CASE 6-4


The patient presented to Dr. Elhart with complaints of chest discomfort. A 2-D Doppler and color-flow Doppler were performed at the local hospital in the outpatient cardiology department, and Dr. Elhart monitored the echocardiography.




6-4A  Echocardiogram


LOCATION: Outpatient, Hospital


PATIENT: Herbert Gillford


PHYSICIAN: Marvin Elhart, MD


STUDY: The study is 2-D and a color-flow Doppler echocardiography.


INDICATION FOR STUDY: Sinus bradycardia


Chamber dimension by M-mode:



DOPPLER:



2-D ECHO (ECHOCARDIOGRAM):



CONCLUSION:



RECOMMENDATIONS:





Case 6-4A  Discussion


This patient was referred for the symptom of sinus bradycardia (R00.1/427.89); but there are more definitive diagnoses available based on the test results. In the Conclusion section of the report, the physician indicates “left ventricular enlargement with severe global left ventricular systolic dysfunction due to valvular insufficiency.” The ventricular enlargement and dysfunction are “due to” another more specific condition (valves are not functioning properly) and therefore neither the enlargement nor the dysfunction are reported separately. The physician indicated both aortic and mitral insufficiency and, in the Doppler section of the report, he indicated tricuspid and pulmonary insufficiency. No specific cause for these conditions was specified, so each of the conditions must be reported.


ICD-10-CM: There are two codes required to report these diagnoses. In the Index of the ICD-10-CM, reference the main term “Insufficiency” and subterms “mitral, with, aortic valve disease”, which is coded to I08.0; however, “tricuspid [valve] disease” is also documented. A combination code describing combined rheumatic disorders of mitral, aortic, and tricuspid valves is assigned. Although documentation does not indicate that the multiple valve diseases are rheumatic in nature, the “Includes” note under category I08 states that this category also includes multiple valve diseases that are unspecified, therefore, I08.3 is assigned. In the “Doppler” section of the report, the physician indicates that the patient also has tricuspid and pulmonary insufficiencies. The pulmonary valve insufficiency is reported with I08.8.


ICD-9-CM: There are three codes required to report these diagnoses—one code for each of the insufficiencies. In the Index of the ICD-9-CM, reference the main term “Insufficiency” with subterms “mitral, with aortic valve disease” to be directed to the Tabular to reference 396.3. Reference “Insufficiency, tricuspid” to be directed to refer to the Tabular code entry 397.0. The Index for “Insufficiency, pulmonary, valve” refers you to 424.3. The Tabular for 424.3 indicates “insufficiency NOS” and “Excludes that specified as rheumatic (397.1)”, indicating this is the correct code to assign to this pulmonary valve insufficiency.



Holter


Another commonly used cardiac diagnostic tool is the Holter monitor, which is a portable electrocardiography device that is worn by the patient, usually for 24 hours but up to 48 hours. The monitor records the electrical function of the heart and allows the physician to analyze the data to diagnose various cardiac conditions.


The sequence of events is important to know before you code the Holter monitor service. When the patient’s physician evaluates the patient and determines that the patient requires the monitoring of cardiac function by means of a Holter monitor, the physician orders the monitoring, and at a later time the patient presents to the cardiology laboratory to be fitted with a monitor. The technician, under the supervision of the cardiologist, outfits the patient with the monitor, and the patient leaves the department wearing the monitor for a specified period of time (usually 24 to 48 hours for a Holter). The patient returns to the cardiology department, and the technician removes the monitor. The cardiologist interprets tracing of the Holter and writes a report of findings that is sent to the physician who ordered the monitoring. In the meantime, after the monitoring has been performed, but before the results are returned and reviewed by the physician, the coder reports the services. This sequence is important in identifying the diagnosis for which the services were provided. The coder does not wait for the results of the various tests to be returned before reporting the services for reimbursement. The coder does, however, wait for the test to be conducted prior to submitting for reimbursement. The physician’s diagnosis will be used to report the services for the Holter monitoring in the following report. After the physician reads the report, he or she may change the diagnosis of the patient to reflect the test results, but only the physician can make the determination of the diagnosis. Perhaps the physician ordered several tests and the interpretation as to the diagnosis varied. It is the ordering physician’s responsibility to assess all test results and make the conclusion as to the final diagnosis. When coding the various reports in this text, you will be seeing the report results. However, you would usually only know that the test had been requested and performed, not the results.



CASE 6-5   6-5A Holter Report 6-5B Radiology Report, Preimplantation 6-5C Operative Report, Pacemaker Implantation 6-5D Radiology Report, Postimplantation


CASE 6-5




6-5A  Holter report


Code the following Holter service, assuming the test had been ordered and performed. Report the global service. For the diagnosis, you would use the information presented in the “Indication” section of the report, because that would be the reason the physician ordered the test.


LOCATION: Outpatient, Clinic


PATIENT: Herbert Gillford


PHYSICIAN: Dr. Marvin Elhart


INDICATION: Patient with atrial fibrillation on Lanoxin. Patient has known cardiomyopathy.


BASELINE DATA: An 86-year-old man with congestive heart failure who is taking Elavil, Vasotec, Lanoxin, and Lasix.


The patient was monitored for 24 hours during which time the analysis was performed.


INTERPRETATION:



CONCLUSION:





6-5B  Radiology report, preimplantation


Code the preimplantation services.


LOCATION: Outpatient, Hospital


PATIENT: Herbert Gillford


PHYSICIAN: Dr. Marvin Elhart


RADIOLOGIST: Dr. Morton Monson


EXAMINATION OF: Chest


CLINICAL SYMPTOMS: Sick sinus syndrome


CHEST, TWO VIEWS, FRONTAL AND LATERAL, 11:00 am: Comparison is made to films taken 3 years ago. There is cardiomegaly. Overt failure is not identified. There is only a moderate degree of inspiration. Osseous structures show old compression deformity of the lower thoracic spine. Calcification is identified within a tortuous aorta. The portion of the abdomen that was seen is unremarkable.


IMPRESSION:





6-5C  Operative report, pacemaker implantation


LOCATION: Outpatient, Hospital


PATIENT: Herbert Gillford


SURGEON: Marvin Elhart, MD


PROCEDURE PERFORMED: Dual-chamber pacemaker implantation


INDICATION: Bradyarrhythmia


BRIEF HISTORY: This patient has been experiencing recurrent syncope. He was evaluated in the last year or so. Because of the presence of first-degree AV (atrioventricular) block and sinus bradycardia, the cause for his syncope is his bradyarrhythmia; for that reason, a dual-chamber pacemaker implantation was recommended after discussion with his cousin, who consented to the procedure. The cousin was informed of all potential complications, including infection, hematoma, pneumothorax, hemothorax, myocardial infarction, and even death. He agreed to proceed.


PROCEDURE: The patient was brought to the cardiac catheterization laboratory. He was placed on the catheterization table, where he was prepped and draped in the usual fashion. The procedure was extremely difficult to perform as a result of the patient’s agitation despite adequate sedation. With reasonable hemostasis, the pacemaker pocket was performed in the left infraclavicular area after anesthetizing the area with 0.5 cc (cubic centimeter) of Xylocaine. Hemostasis was secured with cautery. The patient had excessive venous oozing from Valsalva and straining, and that was controlled with pressure. A single stick was performed because of the patient’s agitation. Using a 9-French peel-away sheath, we introduced an atrial and a ventricular lead and placed them in an excellent position.


Thresholds were obtained adequately. The leads were sutured using 0 silk over their sleeves and secured. The pulse generator was connected. The pacemaker pocket was flushed with antibiotic solution. The pacemaker and leads were placed in the pocket and the pocket closed in two layers.


COMPLICATION: None


EQUIPMENT USED: Pulse generator was Medtronic model 60 Thera DRI, serial B28H. The ventricular lead was Medtronic serial L420V, model 4524 Link. The atrial lead was Medtronic 24-58, serial 326V.


The following parameters were obtained after implantation: Pacing threshold in the atrium was excellent at 0.5 msec and 0.5 V, and impedance was 445 ohms and sensing 2.1 mV. In the ventricle, 0.5 msec and 0.3 V with R wave of 19.9 mV and impedance 668 (device evaluation).


The following parameters were left at implantation: DDDR with lower rate limit of 70 and an upper rate limit of 120. The amplitude was 3.5 V in the atrium at 0.4 msec with a sensitivity of 0.5 mV. The ventricle was 3.5 V and 0.4 msec at 2.8-mV sensitivity (device evaluation).


CONCLUSION: Successful implantation of dual-chamber pacemaker without immediate complications.


PLAN: Patient to return to recovery unit and to be discharged late this evening to the nursing home with routine postpacemaker care.




6-5D  Radiology report, postimplantation


Two diagnoses are reported with this postimplantation service. The first listed diagnosis is a Z/V code that indicates “Status, post, pacemaker, cardiac.” The Z/V code is listed first, as that is the reason for the service (checking on the placement of the pacemaker). The bradycardia is also reported, but sequenced after the Z/V code.


LOCATION: Outpatient, Hospital


PATIENT: Herbert Gillford


PHYSICIAN: Dr. Marvin Elhart


RADIOLOGIST: Dr. Morton Monson


EXAMINATION OF: Chest


CLINICAL SYMPTOMS: Status post pacemaker placement, bradycardia


CHEST, SINGLE VIEW, FRONTAL: FINDINGS: This examination is compared with an examination performed earlier on the same day at 11:00 am. Since the previous examination, there has been interval placement of a pacemaker. One of the leads overlies the expected location of the right ventricle. The proximal lead overlies the expected location of the right atrium near the junction with the superior vena cava. Cardiomegaly is present on this examination. This is unchanged when compared with the previous examination. The pulmonary vascular markings appear within normal limits. The examination otherwise appears unchanged compared with the previous examination. Definite pneumothorax is not identified on this examination.





Stress tests


A cardiovascular stress test is a special type of echocardiogram that compares the electrical system of the heart at rest and under exertion. The test is used by the physician to diagnose various diseases, such as coronary artery disease (atherosclerosis), coronary ischemia (dead or dying heart muscle), and arrhythmias (irregular heartbeats). The technician administers the stress test under the direct supervision of the physician. Usually the test is conducted while the patient is exercised on a treadmill or bicycle and continuous recordings are made of the electrical activity of the heart. Electrodes are attached to the chest of the patient, as is a blood pressure monitor. The patient begins to exercise at a low speed. The speed is increased at set intervals until the patient’s maximal or submaximal heart rate is reached and sustained. Three services are bundled into the complete stress test procedure as described in 93015:



Codes in the range 93016-93018 are used when only a component of the test is provided:



In order to report the correct code selection, place of service must be considered. If a physician performs continuous monitoring, supervises the stress test, interprets the data, and compiles a report in an office or physician clinic setting, the entire service would be coded as 93015 without any modifier to show that the global service is being reported to include both the professional and technical components. This will indicate that the physician not only provided the service but also owns the equipment and only one entity will be submitting a charge. Often the stress test is performed in a hospital setting, which will require that the physician and facility each report their distinct component codes without appending any modifiers, since specific codes are provided that describe their respective services. When the physician provides supervision of the stress test but without the interpretation and report, only 93016 would be reported. Code 93018 would be reported when the physician provides the supervision, interpretation, and report. When the physician reports either 93016 or 93018, the facility would report the technical component for the stress test tracing with code 93017.


Sometimes, as the result of physical limitations, a patient is unable to exercise on a treadmill or bicycle. In these instances, the patient can be administered a drug, such as Persantine, adenosine, Cardiolite, or dobutamine to mimic the stress to the heart that would be brought about by exercise by dilating the vessels. These agents are administered in the outpatient setting at the hospital, and the hospital would report the cost of the pharmacologic agent, not the physician. Where tests are performed and who performed each of the components will have an effect on how the services are coded. For instance, if the test was administered at the local hospital and the physician did the supervision only, he would report the supervision component with 93016. The hospital would report the tracing portion of the service with 93017. If the outpatient facility employed a physician to do the supervision and interpretation and a technician to do the tracing, you would report the complete stress test service with 93015.



CASE 6-6   6-6A Adenosine Cardiolite Stress Test 6-6B Radiology Report, Perfusion Scan 6-6C Echo Doppler Report


CASE 6-6




6-6A  Adenosine cardiolite stress test


The stress test was conducted at the cardiology laboratory at the local hospital with the clinic physician supervising the test and interpreting the results.


LOCATION: Outpatient, Hospital


PATIENT: Eleanor Montgomery


PHYSICIAN: Marvin Elhart, MD


INDICATIONS: Atherosclerotic heart disease with prior myocardial infarction, evaluate for potential ischemia


Electrocardiogram at rest in the exercise lead position reveals the presence of normal sinus rhythm with a somewhat atypical left bundle-branch block with related repolarization abnormalities. Subsequently the patient was injected with 140 μg (microgram) per kilogram per minute of IV (intravenous) Adenoscan over a 6-minute period. At the 3-minute mark, Cardiolite was injected. At the 10-minute mark, the patient was brought for Cardiolite imaging. The patient had a little chest pain and chest tightness during this test that resolved by the end of the Adenoscan infusion. At no point did any diagnostic ST (sinus tachycardia) abnormalities develop beyond baseline.


No ectopy was seen other than one PVC (premature ventricular contraction).


The pulses ranged in the 60s to 70s during this test. Systolic blood pressures ranged in the 130s to 160s during this test.


CONCLUSION:





6-6B  Radiology report, perfusion scan


LOCATION: Outpatient, Hospital


PATIENT: Eleanor Montgomery


PHYSICIAN: Dr. Marvin Elhart


EXAMINATION OF: Stress and rest myocardial perfusion scan with ejection fraction quantifications


INDICATIONS: Arteriosclerosis, coronary vessel


CLINICAL SYMPTOMS: Postmyocardial infarction, 20xx, angiography, 20xx


STRESS AND REST MYOCARDIAL PERFUSION SCAN: TECHNIQUE: Yesterday 31.4 mCi (millicurie) of technetium-99 m sestamibi was administered following stress with adenosine. Today, 24.4 mCi of technetium-99 m sestamibi was administered at rest. SPECT (single photon emission computed tomography) imaging was performed.


FINDINGS: There is minimal thinning of the cardiac apex on the stress images that shows some reversibility at rest. There is also a mild defect involving the inferolateral portion of the left ventricle extending from the periapical portion of the mid-left ventricle. This also shows reversibility in the inferior portion of the left ventricle. Ejection fraction about 35%.


IMPRESSION: Mild myocardial perfusion defects involving the apex and the periapical inferolateral portion of the left ventricle, which show reversibility on rest image. Diagnosis is coronary arteriosclerosis.




6-6C  Echo doppler report


The patient’s chest pains may or may not be related to the aortic and mitral insufficiency, so the chest pain would be reported in addition to the insufficiencies.


LOCATION: Outpatient, Hospital


PATIENT: Eleanor Montgomery


PHYSICIAN: Marvin Elhart, MD


INDICATION: Chest pain; evaluate heart function.


M-Mode, 2-D echo, and Doppler studies with color-flow analysis were performed. Findings are as follows:



1. CHAMBER SIZES: The left ventricle appears to be mildly enlarged on the 2-D images and mildly concentrically hypertrophied except for the septum, which is relatively thin compared with the rest of the left ventricle. The left atrium appears to be mildly enlarged and normal in thickness. The right ventricle and right atrium appear to be of normal size and thickness.


2. WALL MOTION (This is pulsed wave): All cardiac chambers contract normally except for the left ventricular interventricular septum; anterior wall appears to be hypokinetic with moderate impairment of LVEF, visually on the order of about 35% or so.


3. The AORTIC ROOT is normal in size.


4. There is no pericardial effusion.


5. VALVES: The aortic valve and mitral valve leaflets are nonspecifically fibrocalcific. The tricuspid valve and pulmonic valve appear normal. No cardiac valves appear to prolapse.


6. DOPPLER WITH COLOR-FLOW INTERROGATION reveals mild aortic insufficiency. There also appears to be moderate mitral insufficiency. No other valvular, stenotic, or regurgitation lesions are seen.


CONCLUSION: The present echo Doppler study reveals mild aortic insufficiency along with moderate mitral insufficiency. The left ventricle and left atrium appear to be moderately enlarged. (The hypertrophy is a result of the insufficiency.) There appears to be left ventricular septal and anterior-wall hypokinesis along with moderate impairment of LVEF in the order of about 35% or so. Please see above report for details.


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May 17, 2017 | Posted by in GENERAL & FAMILY MEDICINE | Comments Off on Cardiovascular system

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