When should a diabetic patient be initially screened for diabetic retinopathy?
It is currently estimated that as of 2014, only about 60% of diabetics have a yearly screening for diabetic retinopathy. Currently, screening is recommended for type 1 diabetics 5 years after the onset of their disease. Type 2 diabetics should be screened at diagnosis and yearly thereafter.
The duration of diabetes is a major risk factor in the development of diabetic retinopathy. After 5 years, approximately 25% of type 1 patients have retinopathy. After 10 years, 60% develop retinopathy, and after 15 years, 80% will be affected.
Of type 2 patients over the age of 30 who have had diabetes for less than 5 years, 40% of those taking insulin and 24% of those not taking insulin have retinopathy. The percentages increase to 84% and 53%, respectively, when the duration has been documented for up to 19 years.
Currently, the gold standard imaging for the diagnosis and classification of diabetic retinopathy is stereoscopic color photographs using seven standard fields. However, this is very labor intensive. There is evidence that single-field fundus photographs in the hands of trained readers can serve as an effective screening tool.
The Snellen best-corrected distance visual acuity is 20/150 in the right eye and 20/50 in the left eye as measured with his glasses. A refraction done in the office does not improve his vision. His pupils are equally round and reactive to light. There is no relative afferent pupillary defect in either eye. Extraocular movements are full. The intraocular pressures are 14 and 17 in the right and left eye, respectively.
On slit lamp exam of his eyes, there are no eyelid or adnexal abnormalities. His conjunctiva and sclera are noninjected and both corneas are clear. The anterior chambers are deep without evidence of cell, flare, or blood. He has lightly pigmented, hazel irises. On careful inspection, there are no abnormal blood vessels present on either iris.
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Clinical Pearl
Iris neovascularization, or rubeosis iridis, is a sign of ocular ischemia and has a variety of etiologies, including diabetic retinopathy. It is most commonly seen at the border of the pupil. It is clinically significant because it may be an indicator of neovascularization of the anatomic angle of the eye. The drainage outflow for aqueous humor is found in the angle, and these vessels may contract to seal it off, resulting in a secondary angle-closure attack.
For dilation, one drop of tropicamide 1% and one drop of phenylephrine 2.5% are instilled into both eyes. The lenses both have cataract changes, including central posterior subcapsular cataracts.